Professor Tobias Langenhan
Head of Department of General Biochemistry, Medical Faculty, University of Leipzig, Germany
Tobias completed his medical studies at the University of Würzburg Medical School, Germany, in 2004, and obtained a research-based Dr. med. degree on the functional neuroanatomy of the olfactory system in 2006, also from Würzburg University. Funded through a Wellcome Trust scholarship he moved to the University of Oxford and Somerville College, Oxford, UK, in 2004 where he studied neuroscience and obtained his M.Sc. in 2005. At Magdalen College he continued his doctoral studies on the role of adhesion GPCRs in neurodevelopment and obtained his D.Phil. from the University of Oxford in 2009. Upon his return to Würzburg University, Germany, he established his own lab and widened his scope to the function of adhesion GPCRs and the principles underlying the molecular architecture of the presynapse. In 2016 he became Heisenberg professor at Würzburg University and in the same year moved to Leipzig University to chair the Department of General Biochemistry at the Rudolf Schönheimer Institute of Biochemistry at the Medical Faculty of Leipzig University.
Tobias is founder, board member and co-chair of the Adhesion GPCR Consortium and since 2014 spokesperson and coordinator of the DFG Research Unit Consortium ‘Elucidation of adhesion GPCR signaling'. He has received several awards including a Wellcome Trust Prize Scholarship (2004), the Wolfgang Bargmann Prize (2006), and a Heisenberg Professorship from the German Research Foundation (2015).
![[Translate to chinese:] Mouse cortical neurons. Transgenic GFP (green). Image courtesy of Prof. Hui Guo, School of Life Sciences, Central South University, China THUNDER_Imager_Mouse_cortical_neuron.jpg](/fileadmin/_processed_/2/a/csm_THUNDER_Imager_Mouse_cortical_neuron_68c961ebdd.jpg "[Translate to chinese:] Mouse cortical neurons. Transgenic GFP (green). Image courtesy of Prof. Hui Guo, School of Life Sciences, Central South University, China")
![[Translate to chinese:] Multiplexed Cell DIVE imaging of Adult Human Alzheimer’s brain tissue section demonstrating expression of markers specific to astrocytes (GFAP, S100B), microglia (TMEM119, IBA1), AD-associated markers (p-Tau217, β-amyloid) and immune cells such as CD11b+, CD163+, CD4+, and HLA-DRA+, clustered around the β-amyloid plaques.](/fileadmin/_processed_/b/b/csm_Alzheimers_brain_tissue_section_showing_astrocytes_microglia_immune_cells_26ab13cde2.jpg "[Translate to chinese:] Multiplexed Cell DIVE imaging of Adult Human Alzheimer’s brain tissue section demonstrating expression of markers specific to astrocytes , microglia , AD-associated markers and immune cells clustered around the β-amyloid plaques")
![[Translate to chinese:] Multiplexed Cell DIVE imaging to characterize the spatial landscape in Human Alzheimer’s Cortical Tissue](/fileadmin/_processed_/7/2/csm_Human_Alzheimers_Cortical_Tissue_Multiplexed_Cell_DIVE_imaging_b053b78344.jpg "[Translate to chinese:] Multiplexed Cell DIVE imaging to characterize the spatial landscape in Human Alzheimer’s Cortical Tissue")
